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10.3390/cells7100156 [DOI] [PMC free article] [PubMed] [Google Scholar] DiGiovanni LF, Khroud PK, Carmichael RE et al (2025) ROS transfer at peroxisome-mitochondria contact regulates mitochondrial redox

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Selenocysteine residues are situated within the active sites of selenoproteins and exhibit superior redox catalytic efficiency relative to cysteine, which contains sulfur rather than selenium [35]

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After treatment, a mean increase of 75.27 mmHg in the PFO was noticed in the HABIT group, which was numerically higher than that of 51.23 mmHg in the control group, but the difference was not statistically significant ( p = 0.113, Fig