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During the early stages of iron overload, a significant amount of released iron is transported to the mitochondria as a buffer, and mitophagy may segregate iron into the mitophagosomes and decrease the origin of ROS for ferroptosis [65, 66]

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Thus, exogenous glutathione may improve vasorelaxation via activation of NO signaling pathway which is known to be impaired with aging

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The coexistence of other thiol-containing biomolecules like cysteine (Cys) and homocysteine (Hcy) in plasma complicates the accurate and selective detection of GSH [89]