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Therefore, considering the important role of mitochondria at all stages of AD, even the pre-MCI stage, mitochondrial dysfunction may play a crucial role as a primary or secondary event in AD, and targeting mitochondrial function may represent promising therapeutic targets

Mutation causes steric hindrance between NA and its inhibitor Framework amino acids (E119, R156, W178, S179, D/N198, I222, E227, H274, E277, N294, and E425) maintain the spatial conformation of enzyme active centers [83,85]

Changes of amino acid serum levels in pediatric patients with higher-risk acute lymphoblastic leukemia (CCG-1961)

Importantly, this mechanism operates independently of the tumor suppressor protein p53, suggesting an alternative pathway for regulating cell proliferation [15]