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Thus, low doses can be neutral from this point of view or induce adaptive responses [24], while exposure to high doses and over the long term affects mitochondrial homeostasis, favoring the generation of ROS, modifying the balance between fission and fusion, altering the function of the respiratory chain, decreasing the mitochondrial membrane potential and triggering apoptosis

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dialysis should not be considered an alternative to NAC

Multiple trials have demonstrated that combination therapy with complementary mechanisms reduces HbA1c more quickly and increases with faster achievement of glycemic targets

Revisiting the steady state kinetic mechanism of glutamine-dependent asparagine synthetase from Escherichia coli