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Conversely, genetic inhibition of autophagy (e.g., Atg7 knockdown) reduced apoptosis and preserved spermatogenesis, suggesting that excessive autophagy can be harmful to germ cells 42

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One proposal suggested that the deglutathionylation activity of glutaredoxins was important for repairing mixed disulfides between glutathione and FeS cluster assembly factors in the oxidizing environment of the mitochondrial matrix [42]

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