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This redox imbalance promotes mitochondrial DNA damage, lipid peroxidation, and activation of apoptotic pathways, ultimately contributing to myofibrillar disorganisation and interstitial fibrosis (73)

doi: 10.3389/fimmu.2024.1421858 Received 23 April 2024 Accepted 23 October 2024 Published 08 November 2024 Volume 15 - 2024 Edited by Bieke Broux, University of Hasselt, Belgium Reviewed by Wei Li, Fudan University, China Zhenhua Li, Harvard Medical School, United States Updates Copyright 2024 Zhang, Lin, Han, Huang, Zhou, Wang, Zhou, Han and Chen

Blood-flow bottleneck BPC-157 (angiogenesis)

Of note, improvements in glycemic control, blood pressure, and lipid profile have been identified, which may lead to a reduction or delay in disease progression

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