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In summary, our data provided compelling evidence demonstrating that Tim-AIII promotes NSCLC ferroptosis by targeting and facilitating HSP90-mediated GPX4 ubiquitination and degradation, which in turn causes cell cycle arrest, inhibition of cell proliferation, and migration

ISSN 0161-5505

Cureus (2022) 14(10):e30603

10.1016/j.neuron.2011.02.006 12 BreunigJ

doi:10.1586/14789450.2014.965159
