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In intact murine islets, blockade of N-type Ca 2+ channels with -conotoxin, but not L-type Ca 2+ channels using nifedipine, abolishes low glucose (1 mM) stimulation of glucagon secretion and blunts the GLP-1-mediated inhibitor effects

The risk of hypoglycaemia would be more relevant if either agent were used alongside insulin or a sulfonylurea, or in the context of severe caloric restriction