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Of the incretin-based therapies, GLP-1 receptor agonists (GLP-1RAs) directly activate receptors, whereas dipeptidyl peptidase 4 (DPP-4) inhibitors exert their action by preventing degradation of endogenous GLP-1 and GIP 7

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In mice, genetic deletion or pharmacologic inhibition of DPP4 is able to increase the homing of CXCR4+ EPC at sites of myocardial damage, resulting in a reduced cardiac remodeling and improved heart function and survival (200)

[DOI] [PMC free article] [PubMed] [Google Scholar] 82.Gault VA, Kerr BD, Harriott P, Flatt PR

doi: 10.3389/fphys.2022.983961 Received 01 July 2022 Accepted 07 September 2022 Published 20 September 2022 Volume 13 - 2022 Edited by Claudio de Lucia, Scientific Clinical Institute Maugeri (ICS Maugeri), Italy Reviewed by Oscar Lorenzo, Health Research Institute Foundacion Jimenez Diaz (IIS-FJD), Spain Raffaele Carraro, La Princesa University Hospital, Spain Updates Copyright 2022 Kreiner, Hovingh and von Scholten