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GLP-1 also suppresses the activation of macrophages and decreases levels of pro-inflammatory markers such as IL-6 and TNF-

Additional evidence derives from retrospective cohort studies reporting intermediate-term mortality outcomes in patients with baseline HF (without detailed phenotyping)

This may influence several physiological functions including, but not restricted to, lowering both pre- and afterload of the heart and downregulation of sympathetic activity, and decreased intraglomerular pressure which is believed to be mediated by increased tubuloglomerular feedback

A significant increase of ANP level after the administration of a GLP-1 agonist was found mainly in experimental models with hyperreninemia and fluid overload-dependent hypertension 8