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Peer review Peer review information Nature Communications thanks Agnieszka Piekielko-Witkowska and the other, anonymous, reviewer(s) for their contribution to the peer review of this work

This concerned not only overt hypothyroid HT patients but also subclinical and euthyroid HT patients

1998, doi: 10.1359/jbmr.1998.13.7.1158

Excessive production of ROS can damage cellular components, including DNA, lipids, and proteins, disrupting the endogenous redox balance and triggering apoptotic mechanisms [40]

The Nitric Oxide Synthase Family of Enzymes Endogenous production of NO occurs through the family of nitric oxide synthase (NOS) enzymes via the oxidation of l-arginine to l-citrulline (Figure 1b) [16]