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Mechanistically, the gK/UL20 protein complex interacts with the viral fusogen gB and this interaction is necessary for gB-mediated membrane fusion during virus entry, as well as virus-induced cell-to-cell fusion and the formation of syncytia 20,21

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[24] In another human clinical trial conducted on a sample of pulmonary tuberculosis patients, epitalon did not appear to correct pre-existing structural aberrations of chromosomes associated with telomere degradation, but did appear to exert a protective effect against the future development of additional chromosomal aberrations