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Although GSH is not alone responsible for these protective mechanisms, the hepatic production of GSH seems to be essential to survival, with knockout mice dying within months secondary to inflammation and hepatic steatosis resulting in liver failure [21]

The bell-shaped Met dimerization profile could be explained by a theoretical receptor-cross-linking model proposed for receptors with bivalent ligands bearing two chemically identical functional groups 32

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[6] [1] This delayed emptying helps moderate post-prandial glucose excursions

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