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ROS are a normal product of cell metabolism and are able to react with cellular components, such as DNA and membrane lipids, contributing to epithelial cell inflammation, airway hyperreactivity, tight junction barrier permeability, and lung injury [74,75]

The Cell Biology of Inflammasomes: Mechanisms of Inflammasome Activation and Regulation

The effect on brain cells may be even greater due to the high lipid membrane content of this organ, and it is conceivable that a brain localized cascade of free radical oxidation stress develops in the brain tissues leading to an encephalitis situation similar to what happens with the cytokine storm and the production of respiratory distress

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These complexities are further underscored by a recent report by Soriani et al., which suggests that CD38 can act as a decoy for extracellular cGAMP, a mediator of innate immunity