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The initiation and progression of AD is characterized by disturbances in intracellular proteostasis due to the alteration of protein synthesis, folding, surveillance, and degradation, which trigger the accumulation of altered proteins and toxic aggregates, widely recognized to be implicated in this phenomenon (327, 330332)

FABPs enhance free fatty acids solubility and facilitate transfer to specific cellular compartments (mitochondria and peroxisomes for oxidation

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