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By genetically deleting the disulfide reductase pathways from mouse liver, we uncovered an inducible, ubiquitous, cell-autonomous reductase-independent backup pathway for the generation of cysteine, driven by cystine CS bond cleavage.

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Studied for its potential to interact with NNMT and influence cellular metabolism in laboratory settings

As pathogenic microorganisms are destroyed (often by an antimicrobial), toxins and lipoproteins are released into the body

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